BREAKING STUDY: SPIKE PROTEIN TRIGGERS PRION-LIKE PROTEIN MISFOLDING, AMYLOID FORMATION, AND MULTI-ORGAN DAMAGE
We found that both COVID “vaccine” spike protein and lab-made SARS-CoV-2 spike protein are potent prion-like drivers of proteostatic collapse, pathological cross-seeding, transcriptional instability, and progressive tissue dysfunction across multiple organ systems.
Spike protein contains intrinsic prion-like and amyloid-forming regions capable of generating abnormal protein aggregates and cross-seeding host proteins including amyloid-β and tau.
Spike protein has been found to persist in humans for at least 3.5 years, extending the period during which abnormal intracellular processing, aggregation, and cross-seeding can occur.
This process may contribute to neurodegenerative pathology, amyloid microclot formation, large white fibrous clots, cardiovascular injury, and progressive multi-organ dysfunction.
Those responsible for unleashing synthetic spike protein on the entire global population must be held accountable.
